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hepatic acp levels  (Elabscience Biotechnology)


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    Elabscience Biotechnology hepatic acp levels
    Effect of CTE on liver <t>ACP</t> <t>levels</t> in DM and Dyslipidemia Rat Model. (a) ACP level (U/L); (b) ACP level (U/mg protein) * The data are presented as means±SD and were obtained from four repetitions. The experimental groups: Group I: Negative control (NC) comprising normal rats, Group II: Positive control (PC) consisting of rats with DM and dyslipidemia, Group III: PC+CTE 200 mg/kg BW (CTE200), Group IV: PC+CTE 400 mg/kg BW (CTE400), Group V: PC+CTE 800 mg/kg BW (CTE800), Group VI: PC+Simvastatin 0.9 mg/kg BW (SV), Group VII: PC+Glibenclamide 0.45 mg/kg BW (GC), and Group VIII: PC+Glibenclamide 0.45 mg/kg BW+Simvastatin 0.9 mg/kg BW (GS). Superscript signs on Figure 5a (a, b, c, cd, d, e) and Figure 5b (a, b, bc, c, d, e) indicate significant differences (p<0.05) among samples obtained from Tukey's HSD.
    Hepatic Acp Levels, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 5 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/hepatic+acp+levels/pmc11053275-60-20-27?v=Elabscience+Biotechnology
    Average 93 stars, based on 5 article reviews
    hepatic acp levels - by Bioz Stars, 2026-07
    93/100 stars

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    1) Product Images from "Antidiabetic and hepatoprotection effect of butterfly pea flower ( Clitoria ternatea L.) through antioxidant, anti-inflammatory, lower LDH, ACP, AST, and ALT on diabetes mellitus and dyslipidemia rat"

    Article Title: Antidiabetic and hepatoprotection effect of butterfly pea flower ( Clitoria ternatea L.) through antioxidant, anti-inflammatory, lower LDH, ACP, AST, and ALT on diabetes mellitus and dyslipidemia rat

    Journal: Heliyon

    doi: 10.1016/j.heliyon.2024.e29812

    Effect of CTE on liver ACP levels in DM and Dyslipidemia Rat Model. (a) ACP level (U/L); (b) ACP level (U/mg protein) * The data are presented as means±SD and were obtained from four repetitions. The experimental groups: Group I: Negative control (NC) comprising normal rats, Group II: Positive control (PC) consisting of rats with DM and dyslipidemia, Group III: PC+CTE 200 mg/kg BW (CTE200), Group IV: PC+CTE 400 mg/kg BW (CTE400), Group V: PC+CTE 800 mg/kg BW (CTE800), Group VI: PC+Simvastatin 0.9 mg/kg BW (SV), Group VII: PC+Glibenclamide 0.45 mg/kg BW (GC), and Group VIII: PC+Glibenclamide 0.45 mg/kg BW+Simvastatin 0.9 mg/kg BW (GS). Superscript signs on Figure 5a (a, b, c, cd, d, e) and Figure 5b (a, b, bc, c, d, e) indicate significant differences (p<0.05) among samples obtained from Tukey's HSD.
    Figure Legend Snippet: Effect of CTE on liver ACP levels in DM and Dyslipidemia Rat Model. (a) ACP level (U/L); (b) ACP level (U/mg protein) * The data are presented as means±SD and were obtained from four repetitions. The experimental groups: Group I: Negative control (NC) comprising normal rats, Group II: Positive control (PC) consisting of rats with DM and dyslipidemia, Group III: PC+CTE 200 mg/kg BW (CTE200), Group IV: PC+CTE 400 mg/kg BW (CTE400), Group V: PC+CTE 800 mg/kg BW (CTE800), Group VI: PC+Simvastatin 0.9 mg/kg BW (SV), Group VII: PC+Glibenclamide 0.45 mg/kg BW (GC), and Group VIII: PC+Glibenclamide 0.45 mg/kg BW+Simvastatin 0.9 mg/kg BW (GS). Superscript signs on Figure 5a (a, b, c, cd, d, e) and Figure 5b (a, b, bc, c, d, e) indicate significant differences (p<0.05) among samples obtained from Tukey's HSD.

    Techniques Used: Negative Control, Positive Control

    Proposed mechanism on how CTE influence metabolic syndrome rats' model * CTE is believed to have a hepatoprotective effect with phenolics and flavonoids content playing a crucial part in scavenging elevated free radicals. Also, flavonoids in CTE have shown the ability to increase the activity of antioxidant enzymes such as CAT and SOD. By elevating the activity of these enzymes, damage pancreatic β cells can be prevented, helping to maintain normal insulin levels in the body. CTE combats oxidative stress by countering free radicals, thereby protecting cells and tissues from damage. Consequently, levels of ACP and LDH, which indicate cell death and cellular injury, are reduced, suggesting decreased injury to the liver and an elevation in the level of liver protein. Additionally, the antioxidant anthocyanins and saponins present in CTE can further contribute to reducing the level of MDA, another indicator of oxidative stress. CTE also can suppress the pro-inflammatory cytokine IL−1β and reduce the levels of CRP, a protein that increases during inflammation, by inhibiting secondary enzymes.
    Figure Legend Snippet: Proposed mechanism on how CTE influence metabolic syndrome rats' model * CTE is believed to have a hepatoprotective effect with phenolics and flavonoids content playing a crucial part in scavenging elevated free radicals. Also, flavonoids in CTE have shown the ability to increase the activity of antioxidant enzymes such as CAT and SOD. By elevating the activity of these enzymes, damage pancreatic β cells can be prevented, helping to maintain normal insulin levels in the body. CTE combats oxidative stress by countering free radicals, thereby protecting cells and tissues from damage. Consequently, levels of ACP and LDH, which indicate cell death and cellular injury, are reduced, suggesting decreased injury to the liver and an elevation in the level of liver protein. Additionally, the antioxidant anthocyanins and saponins present in CTE can further contribute to reducing the level of MDA, another indicator of oxidative stress. CTE also can suppress the pro-inflammatory cytokine IL−1β and reduce the levels of CRP, a protein that increases during inflammation, by inhibiting secondary enzymes.

    Techniques Used: Activity Assay



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    hepatic acp levels  (Elabscience Biotechnology)
    93
    Elabscience Biotechnology hepatic acp levels
    Effect of CTE on liver <t>ACP</t> <t>levels</t> in DM and Dyslipidemia Rat Model. (a) ACP level (U/L); (b) ACP level (U/mg protein) * The data are presented as means±SD and were obtained from four repetitions. The experimental groups: Group I: Negative control (NC) comprising normal rats, Group II: Positive control (PC) consisting of rats with DM and dyslipidemia, Group III: PC+CTE 200 mg/kg BW (CTE200), Group IV: PC+CTE 400 mg/kg BW (CTE400), Group V: PC+CTE 800 mg/kg BW (CTE800), Group VI: PC+Simvastatin 0.9 mg/kg BW (SV), Group VII: PC+Glibenclamide 0.45 mg/kg BW (GC), and Group VIII: PC+Glibenclamide 0.45 mg/kg BW+Simvastatin 0.9 mg/kg BW (GS). Superscript signs on Figure 5a (a, b, c, cd, d, e) and Figure 5b (a, b, bc, c, d, e) indicate significant differences (p<0.05) among samples obtained from Tukey's HSD.
    Hepatic Acp Levels, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/hepatic+acp+levels/pmc11053275-60-20-27?v=Elabscience+Biotechnology
    Average 93 stars, based on 1 article reviews
    hepatic acp levels - by Bioz Stars, 2026-07
    93/100 stars
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    Effect of CTE on liver ACP levels in DM and Dyslipidemia Rat Model. (a) ACP level (U/L); (b) ACP level (U/mg protein) * The data are presented as means±SD and were obtained from four repetitions. The experimental groups: Group I: Negative control (NC) comprising normal rats, Group II: Positive control (PC) consisting of rats with DM and dyslipidemia, Group III: PC+CTE 200 mg/kg BW (CTE200), Group IV: PC+CTE 400 mg/kg BW (CTE400), Group V: PC+CTE 800 mg/kg BW (CTE800), Group VI: PC+Simvastatin 0.9 mg/kg BW (SV), Group VII: PC+Glibenclamide 0.45 mg/kg BW (GC), and Group VIII: PC+Glibenclamide 0.45 mg/kg BW+Simvastatin 0.9 mg/kg BW (GS). Superscript signs on Figure 5a (a, b, c, cd, d, e) and Figure 5b (a, b, bc, c, d, e) indicate significant differences (p<0.05) among samples obtained from Tukey's HSD.

    Journal: Heliyon

    Article Title: Antidiabetic and hepatoprotection effect of butterfly pea flower ( Clitoria ternatea L.) through antioxidant, anti-inflammatory, lower LDH, ACP, AST, and ALT on diabetes mellitus and dyslipidemia rat

    doi: 10.1016/j.heliyon.2024.e29812

    Figure Lengend Snippet: Effect of CTE on liver ACP levels in DM and Dyslipidemia Rat Model. (a) ACP level (U/L); (b) ACP level (U/mg protein) * The data are presented as means±SD and were obtained from four repetitions. The experimental groups: Group I: Negative control (NC) comprising normal rats, Group II: Positive control (PC) consisting of rats with DM and dyslipidemia, Group III: PC+CTE 200 mg/kg BW (CTE200), Group IV: PC+CTE 400 mg/kg BW (CTE400), Group V: PC+CTE 800 mg/kg BW (CTE800), Group VI: PC+Simvastatin 0.9 mg/kg BW (SV), Group VII: PC+Glibenclamide 0.45 mg/kg BW (GC), and Group VIII: PC+Glibenclamide 0.45 mg/kg BW+Simvastatin 0.9 mg/kg BW (GS). Superscript signs on Figure 5a (a, b, c, cd, d, e) and Figure 5b (a, b, bc, c, d, e) indicate significant differences (p<0.05) among samples obtained from Tukey's HSD.

    Article Snippet: The measurements included MDA levels (Elabscience, E-BC-K025-S), hepatic SOD levels (Elabscience, E-BC-K020), CAT levels (Elabscience, E-BC-K031), hepatic LDH levels (E-BC-K046-M), hepatic ACP levels (E-BC-K010-M), hepatic AST levels (Elabscience, E-BC-K236), hepatic ALT levels (Elabscience, E-BC-K235), hepatic IL-1β levels (E-EL-R0012), and hepatic CRP levels (Elabscience, E-EL-R0506) [ , ].

    Techniques: Negative Control, Positive Control

    Proposed mechanism on how CTE influence metabolic syndrome rats' model * CTE is believed to have a hepatoprotective effect with phenolics and flavonoids content playing a crucial part in scavenging elevated free radicals. Also, flavonoids in CTE have shown the ability to increase the activity of antioxidant enzymes such as CAT and SOD. By elevating the activity of these enzymes, damage pancreatic β cells can be prevented, helping to maintain normal insulin levels in the body. CTE combats oxidative stress by countering free radicals, thereby protecting cells and tissues from damage. Consequently, levels of ACP and LDH, which indicate cell death and cellular injury, are reduced, suggesting decreased injury to the liver and an elevation in the level of liver protein. Additionally, the antioxidant anthocyanins and saponins present in CTE can further contribute to reducing the level of MDA, another indicator of oxidative stress. CTE also can suppress the pro-inflammatory cytokine IL−1β and reduce the levels of CRP, a protein that increases during inflammation, by inhibiting secondary enzymes.

    Journal: Heliyon

    Article Title: Antidiabetic and hepatoprotection effect of butterfly pea flower ( Clitoria ternatea L.) through antioxidant, anti-inflammatory, lower LDH, ACP, AST, and ALT on diabetes mellitus and dyslipidemia rat

    doi: 10.1016/j.heliyon.2024.e29812

    Figure Lengend Snippet: Proposed mechanism on how CTE influence metabolic syndrome rats' model * CTE is believed to have a hepatoprotective effect with phenolics and flavonoids content playing a crucial part in scavenging elevated free radicals. Also, flavonoids in CTE have shown the ability to increase the activity of antioxidant enzymes such as CAT and SOD. By elevating the activity of these enzymes, damage pancreatic β cells can be prevented, helping to maintain normal insulin levels in the body. CTE combats oxidative stress by countering free radicals, thereby protecting cells and tissues from damage. Consequently, levels of ACP and LDH, which indicate cell death and cellular injury, are reduced, suggesting decreased injury to the liver and an elevation in the level of liver protein. Additionally, the antioxidant anthocyanins and saponins present in CTE can further contribute to reducing the level of MDA, another indicator of oxidative stress. CTE also can suppress the pro-inflammatory cytokine IL−1β and reduce the levels of CRP, a protein that increases during inflammation, by inhibiting secondary enzymes.

    Article Snippet: The measurements included MDA levels (Elabscience, E-BC-K025-S), hepatic SOD levels (Elabscience, E-BC-K020), CAT levels (Elabscience, E-BC-K031), hepatic LDH levels (E-BC-K046-M), hepatic ACP levels (E-BC-K010-M), hepatic AST levels (Elabscience, E-BC-K236), hepatic ALT levels (Elabscience, E-BC-K235), hepatic IL-1β levels (E-EL-R0012), and hepatic CRP levels (Elabscience, E-EL-R0506) [ , ].

    Techniques: Activity Assay